Cancer of the Ovary - General
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Contents

bulletFIGO Staging
bulletStage I
bulletStage II
bulletStage III
bulletStage IV

FIGO (1988) STAGING (1)

Staging is based on surgicopathological findings.

Stage I:-

bulletGrowth limited to the ovaries.

Stage IA 

Growth limited to one ovary; no ascites; no tumour on the external surface; capsule intact.

Stage IB 

Growth limited to both ovaries; no ascites; no tumour on the external surface; capsule intact.

Stage IC* 

Tumour either stage IA or IB, but with tumour on the surface of one or both ovaries; or with capsule ruptured ; or with ascites present containing malignant cells or with positive peritoneal washings

Stage II:-

bulletGrowth involving one or both ovaries with pelvic extension.

Stage IIA 

Extension and/or metastases to the uterus and/or tubes

Stage IIB 

Extension to other pelvic tissues

Stage IIC

Tumour either stage IIA or IIB, but with tumour on the surface of one or both ovaries; or with capsule(s) ruptured ; or with ascites present containing malignant cells or with positive peritoneal washings

Stage III:-

bulletTumour involving one or both ovaries with peritoneal implants outside the pelvis and/or positive retroperitoneal or inguinal nodes. Superficial liver metastases equals stage III.

Stage IIIA 

Tumour grossly limited to the true pelvis with negative nodes but with histologically confirmed seeding of the abdominal peritoneal surfaces

Stage IIIB 

Tumour involving one or both ovaries with histologically confirmed implants or abdominal peritoneal surfaces none exceeding 2 cm in diameter. Nodes are negative

Stage IIIC 

Abdominal implants greater than 2 cm in diameter and/or positive retroperitoneal or inguinal nodes

Stage IV:-

bulletGrowth involving one or both ovaries with distant metastases. If pleural effusion is present there must be cytology to allot a case to stage IV. Parenchymal liver metastases equals stage IV.

* In order to evaluate the impact on prognosis of the different criteria for allotting cases to stage IC or IIC it would be of value to know if the source of malignant cells detected was peritoneal washings or ascites, and if rupture of the capsule was spontaneous or caused by the surgeon.

Dr James L. Nicklin
M.B., B.S. (Qld), F.R.A.C.O.G., C.G.O
Gynaecologic Oncologist
Queensland Centre for Gynaecological Cancer

 

 

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